well, wikipedia also happens to say this, (i.e. "fail" on above)

Biological analysis and theories
In the 1990s, Dr. Rick Strassman conducted research on the psychedelic drug Dimethyltryptamine (DMT) at the University of New Mexico. Strassman advanced the theory that a massive release of DMT from the pineal gland prior to death or near-death was the cause of the near-death experience phenomenon. Only two of his test subjects reported NDE-like aural or visual hallucinations, although many reported feeling as though they had entered a state similar to the classical NDE. His explanation for this was the possible lack of panic involved in the clinical setting and possible dosage differences between those administered and those encountered in actual NDE cases. All subjects in the study were also very experienced users of DMT and/or other psychedelic/entheogenic agents. Some speculators consider that if subjects without prior knowledge on the effects of DMT had been used during the experiment, it is possible more volunteers would have reported feeling as though they had experienced an NDE.
Dr. Karl Jansen, a New Zealand-born psychiatrist, claims to have reproduced the effects of NDEs through the use of ketamine, thus giving potential evidence of a biological cause of the experience.[46]
Critics have argued that neurobiological models often fail to explain NDEs that result from close brushes with death, where the brain does not actually suffer physical trauma, such as a near-miss automobile accident. Such events may however have neurobiological effects caused by stress.
In a new theory devised by Richard Kinseher in 2006, the knowledge of the Sensory Autonomic System is applied in the NDE phenomenon. His theory states that the experience of looming death is an extremely strange paradox to a living organism - and therefore it will start the NDE: during the NDE, the individual becomes capable of "seeing" the brain performing a scan of the whole episodic memory (even prenatal experiences), in order to find a stored experience which is comparable to the input information of death. All these scanned and retrieved bits of information are permanently evaluated by the actual mind, as it is searching for a coping mechanism out of the potentially fatal situation. Kinseher feels this is the reason why a near-death experience is so unusual.
The theory also states that out-of-body experiences, accompanied with NDEs, are an attempt by the brain to create a mental overview of the situation and the surrounding world. The brain then transforms the input from sense organs and stored experience (knowledge) into a dream-like idea about oneself and the surrounding area.
Whether or not these experiences are hallucinatory, they do have a profound impact on the observer. Many psychologists not necessarily pursuing the paranormal, such as Susan Blackmore, have recognized this. These scientists are not trying to debunk the experience, but are instead searching for biological causes of NDEs.[47]
According to Engmann[48], near-death experiences of people who are clinically dead are psychopathological symptoms caused by a severe malfunction of the brain resulting from the cessation of cerebral blood circulation. An important question is whether it is possible to “translate” the bloomy experiences of the reanimated survivors into psychopathologically basic phenomena, e.g. acoasms, central narrowing of the visual field, autoscopia, visual hallucinations, activation of limbic and memory structures according to Moody’s stages. The symptoms suppose a primary affliction of the occipital and temporal cortices under clinical death. This basis could be congruent with the thesis of pathoclisis – the inclination of special parts of the brain to be the first to be damaged in case of disease, lack of oxygen, or malnutrition – established eighty years ago by C. and O. Vogt.[49] According to that thesis, the basic phenomena should be similar in all patients with near-death experiences. But a crucial problem is to distinguish these basic psychopathological symptoms from the secondary mental associated experiences which may result from a reprocessing of the basic symptoms under the influence of the person’s cultural and religious views.
Some research has suggested that unconscious patients can overhear conversations even if the hospital machines are not registering any brain activity. Research conducted at Sheffield University led to a finding that the release of adrenaline caused by tissue damage during surgery may cause this.[50] Recent findings have also shown that people diagnosed in a "persistent vegetative state" can communicate through their thoughts, as detected by an fMRI.[51][52]


but keep pushing that old time religion.

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"The law, in its majestic equality, forbids the rich as well as the poor to sleep under bridges, to beg in the streets, and to steal bread."